George replied
Monday 11/2-2019 @ kl 13.12 (his time 7:12am on Monday morning)

Dear Allissa,
Thank you for your interest to our project. We will explore your suggestions.
Kindly yours,

I emailed letter to George Tetz
Sunday 10/2-2019 @ kl 18.44

Dr. Tetz,
I listened and watched your interview on Empowering Neurologist, episode 77.

I am excited about your research with the phagobiome, auto-immunity and reading some of the numerous publications. I have thought the same way as you but without a research arm, impossible to test.

Keep going!!! The future is exciting.

Just a few thoughts to leave you with.

Please consider something other than the Vagus Nerve as a pathway of the ENS to the brain. The fascia has been researched as a Sensory Organ by the Fascia Research Society. And the Interstitum, by Dr. Neil Theise, is credited for health of tissue. Bud A.D. Craig, a neuroscientist, found a pathway from the body to the Insula instead of the Somatosensory Cortex in the 1990s. I believe most of this information comes from the fascia, but I have no way to prove this thought.

All my best for future endeavours.
Allissa from Iowa, Living in Sweden

Neil Theise
Neil Theise is a professor of Pathology at the NYU School of Medicine in New York, New York

Rebecca Wells
Rebecca Wells is a professor of Medicine and Bioengineering in the Faculty of the Center for Engineering Mechanobiology at UPenn, part of the University of Pennsylvania in Philadelphia, Pennsylvania.

Bassler Labs working on Quorum Sensing, Fluid Flow and Biofilms.
Bonnie L. Bassler @ Karolinska Institutet “Quorum Sensing and its Control”
Research Lecture at Nobel Forum
Published on Nov 9, 2016
@42:56 – 43:21 We have this fantastic 3-way inter-Kingdom partnership.

Parkinson’s disease and bacteriophages as its overlooked contributors
George Tetz, Stuart M. Brown, Yuhan Hao & Victor Tetz
Scientific Reports
Published: 17 July 2018
volume 8, Article number: 10812 (2018)

•The gut bacteria may be implicated in PD through several pathways, including the effects on the enteric nervous system (ENS) which is in constant direct communication with the brain through the vagus nerve14,15

•The model of gut-originating, inflammation-driven PD pathogenesis is based on the idea that alterations in the intestinal bacterial community may play a role in triggering α-synuclein misfolding in the ENS. According to this model, PD starts in the ENS and then spreads in a retrograde manner through the vagus nerve to the central nervous system16,18,19.

•Moreover, changes in the gut microbiota composition may cause alterations in the intestinal barrier function and permeability, affecting both the immune system and ENS, including neurons and glial cells, and exerting a profound effect on the condition of PD patients21,22,23. Increased intestinal permeability is also associated with activation of enteric neurons and enteric glial may contribute to the initiation of alpha-synuclein misfolding17.